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Browsing Dahili Tıp Bilimleri / Internal Medıcal Scıences by Author "Zeynep Ozozen Ayas"

Now showing items 1-6 of 6

A Rare Epilepsy Type: Eating Epilepsy

Zeynep Ozozen Ayas; Bölük, Ayhan (KARE PUBL, 2017)

Eating epilepsy is a rare type of reflex epilepsy. Genetic factors, types of food, and eating habits are important. Presently described is case of a 28-year-old man with simple partial and secondary generalized epilepsy, ...
Autosomal Recessive Parkinson's Disease with Early-Onset in a Turkish Family

Zeynep Ozozen Ayas; Kotan Dündar, Dilcan; Asli Aksoy Gundogdu (GAZI UNIV, FAC MED, 2019)

Parkinson disease is a progressive, neurodegenerative disease with an increasing incidence of age. It is thought that genetic factors in etiology may be the underlying cause together with environmental factors. Sporadic ...
Cerebral Sinus Venous Thrombosis: Evaluation of Nineteen Patients

Zeynep Ozozen Ayas; Ruhsen Oncel Ocal; Bölük, Ayhan (GALENOS YAYINCILIK, 2018)

Conclusion: Pregnancy and postpartum period are significant risk factors for CSVT. The association of more than one reason in the etiological investigations of patients despite the presence of one significant risk factor ...
Fabry Disease Diagnosis in a Young Stroke Patient: A Case Report

Asli Aksoy Gundogdu; Kotan Dündar, Dilcan; Murat Alemdar; Zeynep Ozozen Ayas (AVES, 2018)
Serum Prolidase Enzyme Activity Level: Not a Predictive Biomarker for Epilepsy

Zeynep Ozozen Ayas; Kotan Dündar, Dilcan; Akdoğan, Mehmet; Günel, Mustafa Ercan (AVES, 2019)

Conclusion: Our study results suggest that although PEA is an enzyme associated with OS and inflammation, it is still not an ideal biomarker for epileptic patients. This study is important because it investigated PEA in ...
The spectrum of neurological manifestations of Fabry disease in alarge Turkish family with c.[680G > A] p.[R227Q]mutation

Asli Aksoy Gundogdu; Zeynep Ozozen Ayas; Murat Alemdar; Kotan Dündar, Dilcan (ASEAN NEUROLOGICAL ASSOC, 2017)

Conclusion: The clinical and genetic features of this large Turkish family with FD support an association between the neurological phenotype and the c.[680G> A] p.[R227Q] mutation. Since FD is treatable, it is recommended ...