Chronic Escitalopram Treatment Induces Erectile Dysfunction by Decreasing Nitric Oxide Bioavailability Mediated by Increased Nicotinamide Adenine Dinucleotide Phosphate Oxidase Activity and Reactive Oxygen Species Production

Modar Kassan; George F. Lasker; Suresh C. Sikka; Sree Harsha Mandava; Ahmet Gokce; Gökçe, Ahmet; Khalid Matrougui; Wayne J. G. Hellstrom; Philip J. Kadowitz; Ege Can Serefoglu

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dc.contributor.authors	Kassan, M; Lasker, GF; Sikka, SC; Mandava, SH; Gokce, A; Matrougui, K; Hellstrom, WJG; Kadowitz, PJ; Serefoglu, EC;
dc.date.accessioned	2020-01-17T11:59:09Z
dc.date.available	2020-01-17T11:59:09Z
dc.date.issued	2013
dc.identifier.citation	Kassan, M; Lasker, GF; Sikka, SC; Mandava, SH; Gokce, A; Matrougui, K; Hellstrom, WJG; Kadowitz, PJ; Serefoglu, EC; (2013). Chronic Escitalopram Treatment Induces Erectile Dysfunction by Decreasing Nitric Oxide Bioavailability Mediated by Increased Nicotinamide Adenine Dinucleotide Phosphate Oxidase Activity and Reactive Oxygen Species Production. UROLOGY, 82, -
dc.identifier.issn	0090-4295
dc.identifier.uri	https://hdl.handle.net/20.500.12619/6916
dc.identifier.uri	https://doi.org/10.1016/j.urology.2013.07.037
dc.description.abstract	CONCLUSION The inhibitory effects of escitalopram on erectile and vascular function were not accompanied by a change in endothelial nitric oxide synthase, neuronal nitric oxide synthase, inducible nitric oxide synthase expression, or endothelial nitric oxide synthase activity, suggesting that the inhibitory effect is caused by a decrease in nitric oxide bioavailability mediated by increased NADPH oxidase and reactive oxygen species production. (C) 2013 Elsevier Inc.
dc.language	English
dc.publisher	ELSEVIER SCIENCE INC
dc.title	Chronic Escitalopram Treatment Induces Erectile Dysfunction by Decreasing Nitric Oxide Bioavailability Mediated by Increased Nicotinamide Adenine Dinucleotide Phosphate Oxidase Activity and Reactive Oxygen Species Production
dc.type	Article
dc.identifier.volume	82
dc.contributor.department	Sakarya Üniversitesi/Tıp Fakültesi/Cerrahi Tıp Bilimleri Bölümü
dc.contributor.saüauthor	Gökçe, Ahmet
dc.relation.journal	UROLOGY
dc.identifier.wos	WOS:000326153300064
dc.identifier.doi	10.1016/j.urology.2013.07.037
dc.contributor.author	Modar Kassan
dc.contributor.author	George F. Lasker
dc.contributor.author	Suresh C. Sikka
dc.contributor.author	Sree Harsha Mandava
dc.contributor.author	Ahmet Gokce
dc.contributor.author	Gökçe, Ahmet
dc.contributor.author	Khalid Matrougui
dc.contributor.author	Wayne J. G. Hellstrom
dc.contributor.author	Philip J. Kadowitz
dc.contributor.author	Ege Can Serefoglu